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Gorillas at Bristol Zoo have spotted evidence of gorillas cheating in a human-like manner. The human-like behaviour was observed as part of a game dreamt up by scientists studied the behaviour of the apes.

Gorillas were presented with a wall-mounted device where the aim is to guide a peanut through a series of obstacles by poking a stick through various holes to move it along.

Gorillas at a zoo in England have demonstrated a distinctly human trait while attempting to solve a new puzzle game - cheating stock. Since first being introduced to the prototype device earlier this year, the scientists say the game has proved a hit with the troop of endangered western lowland gorillas, who regularly returned to play with the game even when there were no more nuts to win.

The prototype device had to be strong enough to withstand a frustrated gorilla, which can be seven times stronger than humans.

Gorillas were presented with a wall-mounted device that spat out a peanut The animals were seen trying to suck out extra peanuts.

Since first being introduced to the prototype device earlier this year, the scientists say the game has proved a hit with the troop of endangered western lowland gorillas.

Most children surpass the intelligence levels of chimpanzees before they reach four years old. A study conducted by Australian researchers in June tested children for foresight, which is said to distinguish humans from animals.

The experiment saw researchers drop a grape through the top of a vertical plastic Y-tube. They then monitored the reactions of a child and chimpanzee in their efforts to grab the grape at the other end, before it hit the floor.

Because there were two possible ways the grape could exit the pipe, researchers looked at the strategies the children and chimpanzees used to predict where the grape would go.

The apes and the two-year-olds only covered a single hole with their hands when tested. But by four years of age, the children had developed to a level where they knew how to forecast the outcome.

They covered the holes with both hands, catching whatever was dropped through every time. The views expressed in the contents above are those of our users and do not necessarily reflect the views of MailOnline.

What Earth might look like in million years: Researchers reveal the four new supercontinents that could form.

Scroll down for video. Share this article Share. Share or comment on this article: He received the Spinoza Prize in and became director of the Hubrecht Institute in In he became one of the national icons of the Netherlands.

Hans Clevers began studying biology at the University of Utrecht in , then began studying medicine as well.

He received an M. For his PhD he studied under Rudy Ballieux. It was there that Clevers cloned the T-cell gene CD3 epsilon.

After his stint at Harvard, he returned to the Netherlands. Describing his path to his career as a medical researcher, Clevers said the following: I actually studied biology first, and then took up medical school at about the same time I was not good with — I liked patients, but I was a little bit impatient with them.

I then decided to go for a post-doc at Boston to Dana Farber, where I really learned the trade. Clevers discovered similarities between the normal renewal of intestinal tissue and the onset of colon cancer.

In he received a grant of two million euros from the Dutch Cancer Society KWF to study the function of stem cells in the normal intestines and in colon cancer, and in and he received ERC Advanced Investigator Grants.

In March , Clevers, who since had been a member of the Royal Netherlands Academy of Arts and Sciences, was elected its president, a position he assumed on 1 June of that year, succeeding Robbert Dijkgraaf.

In connection with his election to this position, he resigned as director from the Hubrecht Institute but kept his research lab there. Since 1 June he is director Research of the Princess Maxima Center for paediatric oncology, located on the Utrecht campus close to the Hubrecht where he maintains his lab.

To summarise his scientific highlights, Hans Clevers identified the crucial downstream component of the Wnt signaling cascade, TCF, and the mechanism by which Wnt signals activate specific TCF target genes.

With these insights and in a collaboration with Bert Vogelstein, he proposed that in APC-deficient colon cancer, it is the inappropriate activation of the Wnt pathway that transforms cells.

He was the first to link Wnt signaling with adult stem cell biology, when he showed that TCF4 gene disruption leads to the abolition of crypt stem cell compartments of the gut.

He went on to show that the Tcf4-driven target gene program in colorectal cancer cells is the malignant counterpart of a physiological crypt stem cell program.

He then described the Wnt target Lgr5 as a marker for adult stem cells including those of crypts. By the creation of several ingenious Lgr5-based transgenic mice, he established the intestinal crypt as one of the pre-eminent models to visualize and study adult stem cells in mammals.

He described several counter-intuitive characteristics for crypt stem cells: Lgr5 stem cells are abundant, they cycle rapidly, they divide symmetrically, and utilize their Paneth cell-daughters as their niche.

Similar results were then reported by him for multiple additional human and mouse tissues. This has opened ways to generate disease models directly from patients as well as avenues for regenerative medicine.

There was a first one, when I just started my lab, within the first few months we cloned the gene that they call TCF1 , T-cell factor 1, I used to be a T-cell embryologist when we first started out.

So in that paper we described cloning of this vector, which at that time maybe on the world scale was not great but for my own lab to clone this gene was my first thing I ever did alone.

When we realized this we started changing our model systems, we used to work on lymphocytes, and we changed it, first to frogs and flies, drosophila, where the Wnt pathway had been studied by many other people that way we could use assays of those people.

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